My positronic brain
There are three things that a woman can not give up:
1. tears
2. cock
3. guilt
not necessarily in the order above.
Saturday, February 27, 2010
Wednesday, February 17, 2010
Long Travel Mini Sandrail Plans
Lessons BIOLOGY
classification scheme animal
1. Sponges (Porifera)
2. Cnidarians (jellyfish)
3. Flatworms (Planaria)
4. Molluscs (snails, clams)
5. Annelids (earthworms)
6. Arachnids (Spiders)
7. Crustaceans (lobsters, crabs)
8. Insects (flies, ants)
9. Echinoderms (sea urchins, starfish)
10. Fish
11. Amphibians (Frogs, Newts)
12. Reptiles (lizards, crocodiles)
13. Birds
14. Mammals
Criteria lessons in powerpoint presentations
not contain many words but only short sentences, diagrams, drawings, photos, links to any videos
Three parts
1. Part One: Enter in schematic form the general structure and the function of the human body
2. Part Two: Getting deeper. Analysis of the structure and function of various organs. Indicate the most common diseases
3. Part III: Comparative Anatomy. Indicate how the apparatus is presented in the various groups of animals show the appearance of adaptive habitat
classification scheme animal
1. Sponges (Porifera)
2. Cnidarians (jellyfish)
3. Flatworms (Planaria)
4. Molluscs (snails, clams)
5. Annelids (earthworms)
6. Arachnids (Spiders)
7. Crustaceans (lobsters, crabs)
8. Insects (flies, ants)
9. Echinoderms (sea urchins, starfish)
10. Fish
11. Amphibians (Frogs, Newts)
12. Reptiles (lizards, crocodiles)
13. Birds
14. Mammals
Criteria lessons in powerpoint presentations
not contain many words but only short sentences, diagrams, drawings, photos, links to any videos
Three parts
1. Part One: Enter in schematic form the general structure and the function of the human body
2. Part Two: Getting deeper. Analysis of the structure and function of various organs. Indicate the most common diseases
3. Part III: Comparative Anatomy. Indicate how the apparatus is presented in the various groups of animals show the appearance of adaptive habitat
What Salary Of Ccie In India
Vitamin D
See also Vitamin D: Vitamin D in foods, vitamin A deficiency D
Vitamin D is needed for bone formation:
deposition of calcium and phosphate in the regions of cartilage;
absorption of calcium (and its phosphate) in the digestive tract;
mobilization of calcium from bone;
renal reabsorption.
CHEMICAL STRUCTURE
The term vitamin D is defined as all compounds with biological activity and are characterized by being calciferol derivatives ciclopentanoperidrofenantrene. The
calciferol (vitamin D3) is the form naturally in mammals, while the ergocalciferol (vitamin D2) is formed upon exposure to ultraviolet light ergosterol (Provitamin form of plant origin). The
calciferol is 50-100 times more active dell'ergocalciferolo (D3 is more active than D2).
Both ergocalciferol that calciferol are inactive forms of vitamin D is therefore needed an activity that occurs in the liver and kidneys.
Man is able to synthesize cholecalciferol from a precursor, which function as provitamin: The dehydrocholesterol (derived from cholesterol reduction). This pro-vitamin is found in skin, so to absorb solar radiant energy that causes the isomerization to cholecalciferol (see synthesis of cutaneous vitamin D). Adequate exposure to sunlight thus reduces the need for vitamin D.
ABSORPTION
The calciferol is absorbed in the intestine of lipids in the same way: enter then become part of the micelles (formed by combination of bile salts with the products resulting from hydrolysis of lipids), is absorbed by diffusion passive in the enterocytes and subsequently incorporated into chylomicrons and transported into the circulation through the mesenteric lymphatic vessels.
Unlike the other fat-soluble vitamins are not stored in the liver calciferol. The amount of calciferol from the diet, however, is very low and the majority of vitamin D is synthesized in the skin by the action of ultraviolet light.
Functions of vitamin D
Vitamin D is essential for the maintenance of calcium and phosphate. The metabolically active form is the 1, 25 - (OH) 2-cholecalciferol which acts by promoting:
calcium absorption in the intestine;
the absorption of calcium and phosphorus in the proximal convoluted tubule;
the deposition of calcium level of bone. The
calciferol acts with a mechanism of action of hormone-because:
is synthesized by the human individual;
acts on a target organ;
has a structure reminiscent of steroid hormones. VITAMIN
DE FOOTBALL
The 1, 25 - (OH) 2-cholecalciferol stimulates the synthesis of CaBP (calcium-carrying protein) in the target organ (enterocytes), intervening at the level of intestinal transcription of DNA encoding the protein and RNA polymerase plasma.
Use dell'actinomicina D and a-amanitin inhibitors of transcription and RNA polymerase, respectively, confirm this action.
This new RNA is synthesized which promotes the synthesis of CaBP necessary to facilitate the absorption of calcium.
It is now certain that this process is involved in the cyclic AMP, which increases in the tissues by the action of vitamin D active.
deficiency and toxicity of vitamin D
The early signs of lack of calciferol are:
reduction of serum calcium and phosphorus
secondary hyperparathyroidism and increased alkaline phosphatase in serum.
signs later are:
inadequate skeletal mineralization (rickets in children, osteomalacia in adults);
muscle weakness, abdominal pain
.
Rickets appears in children between 4 and 24 months of age and is basically inadequate bone mineralization in growth, leading to deformation of the skeleton.
In the first months of life symptoms relate mainly to the skull:
softening in the occipital region, temporal and parietal;
delay in closing the anterior fontanelle (becomes pathological after the fifteenth month of life);
rachitic rosary (between the sixth and twelfth month) hypertrophy of the rib-cartilage junctions;
hypertrophy of the cartilage (in older children) with knots especially wrists and ankles;
bowing of the bones length of the leg and knee valgus.
Osteomalacia occurs in adults with:
muscle weakness;
pain in the back-stretch lumbar spine, pelvic girdle and thighs;
unsteady gait and brittle bones, especially of the spine, shoulders, ribs and basin;
extremely low bone density and the presence of pseudo-fractures, especially the spine, femur and humerus (they radiography);
increased risk of fractures, especially in the hips and wrists. The
hypervitaminosis (excess vitamin D) results in increased intestinal absorption of calcium and bone resorption, hypercalcemia, and decreases serum PTH (amount of parathyroid hormone in the blood, see: calcium and osteoporosis), and loss of calcium homeostasis resulting in:
nausea, vomiting and diarrhea;
hypercalcemia and hypercalciuria, nephrocalcinosis
, cardiocalcinosi and soft tissue calcification.
Excessive intake of calciferol in the diet is extremely unlikely, given the small amount of vitamin D in food, just as you do not know cases of hypervitaminosis due to excessive sun exposure. Instead, you can
intoxication following the administration of calciferol for therapeutic purposes.
food ration made and recommended
Foods rich in vitamin D include:
liver, fish oils, some marine fish (herring, salmon, sardines), smaller amounts are present in eggs, butter and milk.
Almost all vitamin D is synthesized in the skin, so it is recommended that adequate exposure to sunlight, particularly for the elderly.
Under normal exposure to sunlight is sufficient to calciferol meet the needs of the organism, however, especially for safety we recommend the following levels of intake:
infants 10 to 25 g;
children 1 to 3 years 10 g;
children 4 to 10 years 0 to 10 g;
girls 11 ÷ 17 years and boys 0 to 15 g;
adults 0 to 10 g, 10 g
elderly, pregnant
10 g, 10 g
nurse.
1 IU = 0.025 g of 1 g of calciferol calciferol = 40 IU of vitamin D
See also Vitamin D: Vitamin D in foods, vitamin A deficiency D
Vitamin D is needed for bone formation:
deposition of calcium and phosphate in the regions of cartilage;
absorption of calcium (and its phosphate) in the digestive tract;
mobilization of calcium from bone;
renal reabsorption.
CHEMICAL STRUCTURE
The term vitamin D is defined as all compounds with biological activity and are characterized by being calciferol derivatives ciclopentanoperidrofenantrene. The
calciferol (vitamin D3) is the form naturally in mammals, while the ergocalciferol (vitamin D2) is formed upon exposure to ultraviolet light ergosterol (Provitamin form of plant origin). The
calciferol is 50-100 times more active dell'ergocalciferolo (D3 is more active than D2).
Both ergocalciferol that calciferol are inactive forms of vitamin D is therefore needed an activity that occurs in the liver and kidneys.
Man is able to synthesize cholecalciferol from a precursor, which function as provitamin: The dehydrocholesterol (derived from cholesterol reduction). This pro-vitamin is found in skin, so to absorb solar radiant energy that causes the isomerization to cholecalciferol (see synthesis of cutaneous vitamin D). Adequate exposure to sunlight thus reduces the need for vitamin D.
ABSORPTION
The calciferol is absorbed in the intestine of lipids in the same way: enter then become part of the micelles (formed by combination of bile salts with the products resulting from hydrolysis of lipids), is absorbed by diffusion passive in the enterocytes and subsequently incorporated into chylomicrons and transported into the circulation through the mesenteric lymphatic vessels.
Unlike the other fat-soluble vitamins are not stored in the liver calciferol. The amount of calciferol from the diet, however, is very low and the majority of vitamin D is synthesized in the skin by the action of ultraviolet light.
Functions of vitamin D
Vitamin D is essential for the maintenance of calcium and phosphate. The metabolically active form is the 1, 25 - (OH) 2-cholecalciferol which acts by promoting:
calcium absorption in the intestine;
the absorption of calcium and phosphorus in the proximal convoluted tubule;
the deposition of calcium level of bone. The
calciferol acts with a mechanism of action of hormone-because:
is synthesized by the human individual;
acts on a target organ;
has a structure reminiscent of steroid hormones. VITAMIN
DE FOOTBALL
The 1, 25 - (OH) 2-cholecalciferol stimulates the synthesis of CaBP (calcium-carrying protein) in the target organ (enterocytes), intervening at the level of intestinal transcription of DNA encoding the protein and RNA polymerase plasma.
Use dell'actinomicina D and a-amanitin inhibitors of transcription and RNA polymerase, respectively, confirm this action.
This new RNA is synthesized which promotes the synthesis of CaBP necessary to facilitate the absorption of calcium.
It is now certain that this process is involved in the cyclic AMP, which increases in the tissues by the action of vitamin D active.
deficiency and toxicity of vitamin D
The early signs of lack of calciferol are:
reduction of serum calcium and phosphorus
secondary hyperparathyroidism and increased alkaline phosphatase in serum.
signs later are:
inadequate skeletal mineralization (rickets in children, osteomalacia in adults);
muscle weakness, abdominal pain
.
Rickets appears in children between 4 and 24 months of age and is basically inadequate bone mineralization in growth, leading to deformation of the skeleton.
In the first months of life symptoms relate mainly to the skull:
softening in the occipital region, temporal and parietal;
delay in closing the anterior fontanelle (becomes pathological after the fifteenth month of life);
rachitic rosary (between the sixth and twelfth month) hypertrophy of the rib-cartilage junctions;
hypertrophy of the cartilage (in older children) with knots especially wrists and ankles;
bowing of the bones length of the leg and knee valgus.
Osteomalacia occurs in adults with:
muscle weakness;
pain in the back-stretch lumbar spine, pelvic girdle and thighs;
unsteady gait and brittle bones, especially of the spine, shoulders, ribs and basin;
extremely low bone density and the presence of pseudo-fractures, especially the spine, femur and humerus (they radiography);
increased risk of fractures, especially in the hips and wrists. The
hypervitaminosis (excess vitamin D) results in increased intestinal absorption of calcium and bone resorption, hypercalcemia, and decreases serum PTH (amount of parathyroid hormone in the blood, see: calcium and osteoporosis), and loss of calcium homeostasis resulting in:
nausea, vomiting and diarrhea;
hypercalcemia and hypercalciuria, nephrocalcinosis
, cardiocalcinosi and soft tissue calcification.
Excessive intake of calciferol in the diet is extremely unlikely, given the small amount of vitamin D in food, just as you do not know cases of hypervitaminosis due to excessive sun exposure. Instead, you can
intoxication following the administration of calciferol for therapeutic purposes.
food ration made and recommended
Foods rich in vitamin D include:
liver, fish oils, some marine fish (herring, salmon, sardines), smaller amounts are present in eggs, butter and milk.
Almost all vitamin D is synthesized in the skin, so it is recommended that adequate exposure to sunlight, particularly for the elderly.
Under normal exposure to sunlight is sufficient to calciferol meet the needs of the organism, however, especially for safety we recommend the following levels of intake:
infants 10 to 25 g;
children 1 to 3 years 10 g;
children 4 to 10 years 0 to 10 g;
girls 11 ÷ 17 years and boys 0 to 15 g;
adults 0 to 10 g, 10 g
elderly, pregnant
10 g, 10 g
nurse.
1 IU = 0.025 g of 1 g of calciferol calciferol = 40 IU of vitamin D
Tuesday, February 2, 2010
Sports Intimidating Themes
News from 'I'm fucking ...
They gave me selfish.
of nihilism.
And of course I did not miss the classic shit.
But still I missed this: "You were a parenthesis."
What to think of it, I would go too well.
However, for a minimum of intellectual honesty, at least one is specified the type.
Shit, there's a big difference between a square brackets and a brace.
Subscribe to:
Posts (Atom)